Bradycardia-Induced Recurrent Torsade de Pointes: When Serenity Turns into Chaosity
نویسندگان
چکیده
منابع مشابه
Bradycardia-induced Torsade de Pointes – An arrhythmia Less Understood
Torsade de pointes (TdP) denotes a polymorphic ventricular tachycardia that is associated with prolonged QT interval at baseline, and is potentially life-threatening. Though it is well established that the genesis of TdP is closely linked to the transmural dispersion of repolarisation (TDR), the cellular and electrophysiological milieu predisposing to torsadogenesis in many conditions is not co...
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A case of sotalol-induced polymorphous ventricular tachycardia (torsade de pointes) is presented. The patient had moderately prolonged Q-T interval before medication with sotalol with further prolongation after application of this drug. Electrophysiological study during rechallenge with sotalol demonstrated a uniform ventricular tachycardia with a somewhat polymorphous onset; whether or not thi...
متن کاملDrug-induced torsade de pointes.
A73-year-old man with mild coronary artery disease and a dilated cardiomyopathy presented to the emergency room with a hemodynamically stable wide-QRS tachycardia. His 12-lead ECG revealed episodes of ventriculoatrial block, and a diagnosis of ventricular tachycardia (VT) was made (Figure 1). Intravenous procainamide restored sinus rhythm. Tachycardia recurred, and a second bolus of intravenous...
متن کاملMoxifloxacin-induced torsade de pointes
Torsade de pointes (TdP) is increasingly recognized as a complication of drug therapy. The most common cause of drug-induced QT prolongation is inhibition of the rapidly activating component of the delayed potassium current (IKr). Moxifloxacin, a widely used fluoroquinolone, is a weak IKr inhibitor and has been associated with QT prolongation. We report a case of marked QT prolongation (618 ms)...
متن کاملMethadone-induced Torsade de pointes tachycardias.
Methadone is a synthetic opioid frequently used in drug maintenance programs for heroin addicts. It prolongs the QT-interval and is mainly metabolized by the isoenzyme CYP3A4 of the hepatic cytochrome-P450-system, which is used by numerous other QT-prolonging agents. Its most severe side effect is the development of life-threatening Torsade de pointes ventricular tachycardia in the setting of a...
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ژورنال
عنوان ژورنال: International Journal of Cardiovascular Practice
سال: 2018
ISSN: 2476-468X
DOI: 10.21859/ijcp-03045